2015). These studies, taken together, suggest that, While a immediate activation of GPR55 or PPAR‐α takes place, PEA can create its anti‐inflammatory action from the gut also through oblique activation of CB1 and CB2 receptors, possibly due to the power of this compound to potentiate the action of endocannabinoids at these receptors.Palmitoyle
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With this critique, we go over the molecular targets of PEA, its pharmacological exercise in neurological Problems, suffering perception and inflammatory disorders, in addition to the latest proof around the performance of micronized and ultra‐micronized formulations of PEA when specified alone or in combination While using the antioxidant flavon
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